Наукові роботи молодих вчених. Кафедра анатомії людини
Permanent URI for this collectionhttps://repo.knmu.edu.ua/handle/123456789/440
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Item Blood supplying of human’s pericardium(Видавництво ХНМУ, 2013-05) Ізмайлова, Луіза Василівна; Измайлова, Луиза Васильевна; Izmailova, Luiza; Авилова, Ольга Владимировна; Ласкутов, А.В.; Avilova, Olga; Laskutov, A.Item Experience of using simulation-based medical training in Kharkiv National Medical University(2018-10-19) Erokhina, Victoriia; Avilova, OlgaItem Histopathology of the spleen due to exposure of polyethers in experimental study(Charite University Medicine Berlin, 2017-09-27) Avilova, OlgaItem Internationalization of the Ukrainian higher education: problems and prospects(2018-10-19) Erokhina, Victoriia; Avilova, OlgaItem Metastructure of the spleen under the impact of tryglycidyl ether of polyoxypropylene triol(2016-05) Avilova, OlgaItem Microscopic features of the spleen under the impact of xenobiotics(Khatkiv National Medical University, 2018-05-24) Avilova, Olga; John, Chris MathewItem Organometric changes of rat's thymus after influence of propylene glycol(KNMU, 2020-01-20) Ishan, Verma; Avilova, OlgaOne of the global problems of humanity - pollution of environment, which needs immediate solution as adaptation capabilities of human body are not limitless. Xenobiotics impact lead to significant changes in organs and tissues, and subsequently they explicit pathology at the molecular and cellular level, and finally, at the macrolevel. Every year, industrial production introduces dozens of new polyols characterized by significant amounts of synthesis, which carry a potential and real danger to the population and are Aim of the study.Item Osteogenesis imperfecta: wide range of presentation(KNMU, 2020-01-20) Arjun, Batish; Avilova, OlgaIntroduction. Osteogenesis Imperfecta (OI) also known as Brittle Bone Disease is a group of genetic disorders that mainly affects bone. The inheritance commonly follows autosomal dominant pattern or via a new mutation. In 17th century it was called congenital osteomalacia. Prevalence is one in every 15000 live births. There may be in utero death, perinatal death or adult patient can present with complications like cervical artery dissection and aortic dissection. Lack of Type 1 collagen is main underlying pathology. This occurs in more than 90% of cases due to mutations in the COL1A1 or COL1A2 genes. It is classified into eight types based on differences in clinical presentation, bone architecture and diagnosis is based on clinical picture and DNA testing. The newer forms of OI are not associated with type 1 collagen gene defect. Aim of the research. To study history, etiology, pathogenesis, manifestation of OI in relation with collagen: Rare Genetic Disoder.